What are the cognitive theories?

Cognitive theories are characterized by their focus on the idea that how and what people think leads to the arousal of emotions and that certain thoughts and beliefs lead to disturbed emotions and behaviors and others lead to healthy emotions and adaptive behavior.

What neurotransmitter plays a role in anorexia?

In anorexia, the leading hypothesis is that the disorder is associated with an over-production of dopamine, leading to anxiety (Bailer et al., 2012a), harm avoidance (Bailer et al., 2012b), hyperactivity and the ability to go without pleasurable things like food (Kontis & Theochari, 2012).

What is the psychological explanation for anorexia?

Psychological explanations for anorexia nervosa focus on anorexia being caused or triggered by dysfunctional families (e.g. family systems theory); social learning (observation and imitation of role models); and cognitive factors (irrational beliefs and distortions).

What does anorexia do to the nervous system?

Parts of the brain undergo structural changes and abnormal activity during anorexic states. Reduced heart rate, which could deprive the brain of oxygen. Nerve-related conditions including seizures, disordered thinking, and numbness or odd nerve sensations in the hands or feet.

What is the neurobiology of anorexia nervosa?

In anorexia nervosa, severely restricted food intake appears to be related to overactive inhibitory control in combination with underactive reward circuitry. In contrast, dysregulation of both inhibitory and reward drives may manifest in the alternating over- and under-consumption characteristic of bulimia nervosa.

What causes anorexia in the brain?

While bulimics may have low levels of serotonin, other studies indicate that anorexics have high levels of neurotransmitters in some areas of the brain. For example, researchers in London found that anorexics have an overproduction of serotonin, which can cause a continual state of acute stress and anxiety.

What part of the brain is affected by anorexia?

Most fMRI studies performed in patients with anorexia nervosa focused on food, taste, physical appearance and social cognition. Although very different in terms of the study protocol, the most common findings are increased activation of the amygdala and altered activation of the cingulate cortex.

How does classical conditioning explain anorexia?

This may well lead to eating disorders like anorexia. Classical Conditioning suggests that men and women may come to associate thinness (which ought to be a Neutral Stimulus) with glamour and beauty (an Unconditioned Stimulus); this happens because both get the same approval and admiration (the Unconditioned Response).

How does the behaviourist approach explain anorexia?

A cognitive behavioural theory of the maintenance of anorexia nervosa is proposed. It is argued that an extreme need to control eating is the central feature of the disorder, and that in Western societies a tendency to judge self-worth in terms of shape and weight is superimposed on this need for self-control.

What happens to the brain when you have AN eating disorder?

NIH-funded study finds changes can affect food intake control circuitry and cause disorders to progress. Researchers have found that eating disorder behaviors, such as binge-eating, alter the brain’s reward response process and food intake control circuitry, which can reinforce these behaviors.

Is anorexia a neurological disorder?

It is also proposed that bulimia consists of a “positive” neurological subtype and that restricting anorexia represents a “negative” neurological subtype.

Does anorexia cause permanent brain damage?

Brain scans of people with anorexia reveal that the brain goes through structural changes or abnormal activity during the disease. Some of these abnormalities may discontinue weight restoration, but some of the damage to the brain can be permanent.

Does anorexia increase dopamine?

Two months ago, researchers from the University of Pittsburgh reported that recovered anorexics show increased dopamine in the brain. recovered anorexics show increased dopamine in the brain. Dopamine is a chemical involved in weight, feeding behaviors, reinforcement, and reward.

Does bulimia cause low serotonin?

Physiologic and pharmacologic evidence suggest that patients with normal weight bulimia have reduced serotonin activity when acutely ill. Such disturbances, even if secondary to dietary abnormalities, may still contribute to dysphoric mood and binging behavior.

Which eating disorder is most commonly found in males?

Binge Eating Disorder (BED) is the most common eating disorder diagnosis found in males. This disorder involves individuals engaging in bingeing behaviors as one might with bulimia nervosa, however, those struggling with BED do not engage in compensatory behaviors after bingeing.

Which neurotransmitter is associated with eating disorders?

Based on this behavioral neuroscience, it is not surprising that a dysregulation in these feeding-reward-related neurotransmitters and brain areas is seen in studies of eating disorders. Dopamine (blue), serotonin (green), acetylcholine (red) and the opioids (orange) have each been indicated in disordered eating.

How does the brain regulate hunger and eating disorder?

In a non-disordered brain, typically the hypothalamus motivates an individual to eat. In those with an eating disorder, signals from other regions of the brain override the signal in the hypothalamus. This indicates that the brain can reject signals, including taste-reward and hunger [1].

How does being underweight affect the brain?

Being underweight also leads to low haemoglobin count in the blood. Haemoglobin is essential for carrying oxygen to the brain, and being low on this parameter results in poor oxygenation of the brain, leading to regular episodes of dizziness, easy fatigability and cognitive decline.

What is the pathophysiology of anorexia?

Patients with anorexia nervosa have altered brain function and structure there are deficits in neurotransmitters dopamine (eating behavior and reward) and serotonin (impulse control and neuroticism), differential activation of the corticolimbic system (appetite and fear), and diminished activity among the …

Is there a genetic component to anorexia?

Although thought of as a psychological problem, the eating disorder anorexia nervosa often runs in families, suggesting that it has a genetic component. Now researchers have found two genes that help determine the risk of acquiring the disease.

How does anorexia affect the hypothalamus?

The brains of women with eating disorders send signals from other regions that override the hypothalamus.” Anorexia and bulimia causes sufferers to fear eating certain foods – and this study suggests that can ultimately condition the brain to reject signals from the hypothalamus, including taste-reward and hunger …

What are some examples of classical conditioning?

For example, whenever you come home wearing a baseball cap, you take your child to the park to play. So, whenever your child sees you come home with a baseball cap, he is excited because he has associated your baseball cap with a trip to the park. This learning by association is classical conditioning.

Do role models influence anorexia?

In relation to anorexia nervosa, important role models might be mothers, peers or celebrities in the media. Research has shown that mothers who complain about their weight are more likely to have children who have their own weight concerns.

How is classical conditioning nurture?

Classical conditioning (also known as Pavlovian or respondent conditioning) is learning through association and was discovered by Pavlov, a Russian physiologist. In simple terms, two stimuli are linked together to produce a new learned response in a person or animal.

What does the social learning theory say about eating disorders?

Social Learning Theory attributes the etiology of eating disorders to the emphasis placed on thinness in Western society. Women often receive positive attention for being thin, or even for attempting weight loss. This attention then serves as reinforcement of the thin-ideal (Levine & Smolak, 2001).

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